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Like the various different proteins recognized to change tuberin, Ranek discovered protein kinase G altered tuberin by adding phosphates to it, but in a beforehand unidentified region that turned out to supply the sought after brake-like impact. Using genetic engineering tools in coronary heart muscle and connective tissue cells, the workforce mutated human tuberin protein in the particular places that have been altered by protein kinase G.
The alterations made the cells behave in certainly one of two ways: one sort of mutation "turned up" the impact of tuberin all the time while the other basically "turned down" its effect all the time.

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